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G2Voice Broadcast #36: Why so many with Parkinson’s Disease?
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Statistics on Parkinson's


Who Has Parkinson's?
• As many as one million Americans live with Parkinson's disease, which is more than the combined number of people diagnosed with multiple sclerosis, muscular dystrophy and Lou Gehrig's disease.
• Approximately 60,000 Americans are diagnosed with Parkinson's disease each year, and this number does not reflect the thousands of cases that go undetected.
• More than 10 million people worldwide are living with Parkinson's disease.
• Incidence of Parkinson’s increases with age, but an estimated four percent of people with PD are diagnosed before the age of 50.

• Men are one and a half times more likely to have Parkinson's than women.Source:

Parkinson's, ALS, Dementia, Alzheimer's, Multiple Sclerosis, and Epilepsy definitions below.

• Parkinson's: Parkinson's disease affects the nerve cells in the brain that produce dopamine. Parkinson's disease symptoms include muscle rigidity, tremors, and changes in speech and gait. After diagnosis, treatments can help relieve symptoms, but there is no cure.

• Amyotrophic lateral sclerosis (ALS), also known as Lou Gehrig's disease and motor neurone disease
(MND), is a specific disease that causes the death of
neurons which control voluntary muscles. Some also use
the term motor neuron disease for a group of conditions of which ALS is the most common.

Dementia: a chronic or persistent disorder of the mental processes caused by brain disease or injury and marked by memory disorders, personality changes, and impaired reasoning.

Alzheimer’s disease is an irreversible, progressive brain disorder that slowly destroys memory and thinking skills, and eventually the ability to carry out the simplest tasks. In most people with Alzheimer’s, symptoms first appear in their mid-60s. Estimates vary, but experts suggest that more than 5 million Americans may have Alzheimer’s.

Alzheimer's disease is currently ranked as the sixth leading cause of death in the United States, but recent estimates indicate that the disorder may rank third, just behind heart disease and cancer, as a cause of death for older people.

Alzheimer’s is the most common cause of dementia among older adults. Dementia is the loss of cognitive functioning—thinking, remembering, and reasoning—and behavioral abilities to such an extent that it interferes with a person’s daily life and activities. Dementia ranges in severity from the mildest stage, when it is just beginning to affect a person’s functioning, to the most severe stage, when the person must depend completely on others for basic activities of daily living.

Multiple Sclerosis: Multiple sclerosis (MS) is an unpredictable, often disabling disease of the central nervous system that disrupts the flow of information within the brain, and between the brain and body.

The cause of MS is still unknown – scientists believe the disease is triggered by as-yet-unidentified environmental factor in a person who is genetically predisposed to respond.

Epilepsy is a brain disorder in which clusters of nerve cells, or neurons, in the brain sometimes signal abnormally causing strange sensations, emotions, and behavior, or sometimes convulsions, muscle spasms, and loss of consciousness.

We don’t have any Parkinson’s dis-ease testimonies yet, but some of these testimonies below are similar. I have talked with people that have told me their parents either had dementia, Parkinson's or Alzheimer's and it is now cured, but not received a testimony yet. PLEASE, if anyone has a testimony of Parkinson’s disease cured from MMS please, send it to us at: This email address is being protected from spambots. You need JavaScript enabled to view it.. We need it to help the people suffering from this horrible dis-ease of the body. Below are a few testimonies of some Neurological problems that were cured with MMS and the Genesis II Church Sacraments.

Written Testimonies:
Memory came back

An elderly guy (in his 80-90's), who claimed he was the last survivor of the chemical company (since closed), that use to operate in New Plymouth, and made Agent Orange, and many other toxic sprays. After 50 odd years working there, he said that he was so full of toxins that he couldn't remember things that he had done the day before, along with other health effects relating to a build up of toxins in the body. Someone told him about MMS, which he tried. He phoned me on the second day all excited, saying that he remembered all he had done the day before. He claimed to be 100% better within about a week on MMS. He is now an advocate for MMS, telling everyone who is willing to listen.
Posted : 03-02-2015

I have seen many problems solved with MMS. My favorite was when my mother was diognosed with Alzheimers and was given a pill, which after taking the first one at 10 PM, paralized her body but allowed her to talk and she was scared. I listened to her cry when it happened. She did not even know what she was taking. After she told me which bottle, I googled the pills which turned out to be Alzheimers meds and not effective. So my mother when asked, if she wanted to keep taking them, she said no. I gave her 6 drops of the MMS and fifteen minutes later she was fast asleep. In the morning I gave her 6 more drops and an hour later 6 more. By 9:30 AM we had a normal conversation about how she met my father some 65 years before, her clarity of thought had returned. After 1 week of protocol 1000 she felt so good she stopped taking the MMS and after a month her symptoms returned. 1 week on protocol 1000 again she felt great and stopped taking the MMS I tried to get to keep taking it but after a week she felt she had enough sure enough it came back a month later and then we would start again. However the clarity of thought provided my mother was undeniable. 3 years later most of my siblings had forgotten about her alzheimers diognosis.
Keith Pace
PS- Jim Humble you're my Hero.

Alzheimer's gone
I am familiar with Alzheimer's, so when I had symptoms occur, they were familiar to me. I noticed I was not coping socially, and starting to have total gaps in my memory (and thus withdrawing). Because I had heard of mms being used to cure dementia I decided I had nothing to loose. I followed the protocl 1 for one month, slowly building up to the full dose of 3 drops every hour for 8 hours, and staying with the regime, for the full period, (it is important to note that if the dose is causing you to feel unwell, cut back on the doses per day and keep it low for 2 days then start to increase to the level you can tolerate, but do not stop all together). By the end of the treatment I noticed a remarkable change in my cognition, and as time goes on my condition continues to improve. I feel like a new person, I am social again, happy, and look forward to each new day.
Posted : 20-11-2012

My father had a very serious back operation and was under anesthesia for approximately 5 hours. In post-op he was confused. As his stay in the hospital progressed he regressed. He was experiencing significant dementia. The doctors claimed that the surgery just brought forth and underlying problem that was already there. My dad, being 78 at the time was very healthy, ran a ranch, prior to surgery.
One day I got I remembered the MMS1 and decided to give it a try on my dad. By this time he was in assisted care. I did Clare's combination on him and by the time I left that day he was much clearer. I came back the next day and did the same with even greater improvement. By the third day, after the doses, he was back to himself and able to go home.
If not for the MMS I don't think he would of ever been able to leave assisted living. He was getting worse and worse, not knowing where he was or who we were. I was so excited!!! The MMS really works!!
Thanks you for your commitment to help others!
Dianne G



After my mother was diognosed with Alzheimers she hated the effects of the pill so much that she was willing to try MMS. Her symptoms disapeared almost over night the next morning after only drinking 6 drops 3 times she told me the story of how she met my father some 60 years before which was impossable the day before. She continued taking it for a week and then felt so good she stopped taking it within 1 month the symptoms returned after taking MMS again the symptoms went away again. she had to take a daily maintanence dose to keep it at bay.
Thank you Jim Humble you are my Hero,
• Spanish video testimony of MMS curing Alzheimers. Mms cura Alzheimers:
• Spanish video testimony of MMS curing ALS. Testimonio MMS ELA (Esclerosis lateral amiotrófica):
The EXACT cause of Parkinson’s dis-ease is not known according to the Mainstream Medical Industry. One doctor in the video below thinks Alzheimer’s is caused by a bacterium. Could Parlkinson’s be caused by a bacterial infection in the brain? Good question and if so, that is why Chlorine Dioxide kills it because it is anti-bacterial,(bad bacterium).

Doctor talks about Alzheimers being a bacteria casue:

Below is the major consensus of what Parkinson’s is and theories about how it is caused.

What Causes Parkinson's Disease?
Parkinson's disease is caused by the progressive impairment or deterioration of neurons (nerve cells) in an area of the brain known as the substantia nigra. When functioning normally, these neurons produce a vital brain chemical known as dopamine. Dopamine serves as a chemical messenger allowing communication between the substantia nigra and another area of the brain called the corpus striatum. This communication coordinates smooth and balanced muscle movement. A lack of dopamine results in abnormal nerve functioning, causing a loss in the ability to control body movements.


Why Does Parkinson's Disease Occur?
Why Parkinson's disease occurs and how the neurons become impaired is not known. (bold emphasis mine) However, there is increasing evidence that Parkinson's disease may be inherited (passed on genetically from family members).
There is considerable controversy surrounding the possibility of a genetic cause of Parkinson's disease. In a small number of families, specific genetic abnormalities leading to the illness have been identified. However, the vast majority of people with Parkinson's disease do not have one of these identified genetic abnormalities. It is probable that in people who develop Parkinson's disease early in life (called young-onset Parkinson's disease) there is a genetic component. Because we don't understand very much at this point about how Parkinson's disease is inherited, the implications for children of people with Parkinson's disease are unclear.
There is also some evidence that certain toxins in the environment may cause Parkinson's disease. Scientists have suggested that external or internal toxins may selectively destroy the dopaminergic neurons, causing Parkinson's disease. Toxins that may be linked to Parkinson's include manganese, carbon monoxide, carbon disulfide, and some other pesticides.
Also, it is believed that oxidative stress can cause Parkinson's disease. Oxidation is a process in which free radicals (unstable molecules lacking one electron), in an attempt to replace the missing electron, react with other molecules (such as iron). Free radicals are normally formed in the brain and body, but usually the brain and body have mechanisms to get rid of them. In people with Parkinson's disease, the mechanisms may not be effective or they may produce too many free radicals. It is also possible that environmental toxins may contribute to abnormal free radical formation and lead to Parkinson's disease. Oxidation is thought to cause damage to tissues, including neurons. In most cases, antioxidants protect cells from free radical damage.


Are toxins the culprit with when it comes to Parkinson’s disease?


Wednesday December 08, 2010
Viartis - There are now fourteen known toxic causes of Parkinson's Disease : pesticides (Paraquat, Rotenone, Maneb), solvents (Trichloroethylene, Toluene, N-Hexane, Carbon disulfide), MPTP, Mercury, Manganese, Copper, Carbon monoxide, Lead, Cyanide.

Although Agent Orange is widely claimed to be a toxic cause of Parkinson's Disease, not even one study in the entire medical literature has ever shown that to be true.

Toxicity has the potential to be the sole cause of Parkinson's Disease. To very varying extents, toxicity can also be a partial cause of Parkinson's Disease. Symptoms normally develop when toxic exposure occurs or soon after, or gradually increase over time as exposure to toxicity persists.

Symptoms do not develop years or decades after exposure as is often claimed. In fact, there is a tendency for toxic effects to decline over time, but with some toxic substances that can take years.

The prevalence of Parkinson's Disease that is due to toxicity is not known. However, the evidence suggests that the number of people whose Parkinson's Disease is due to toxicity is very low, being the exception rather than the norm.

The toxic causes of Parkinson's Disease each cause different symptoms because of the different biochemical means by which toxicity is caused, and because of the other biochemical functions that each of the particular toxins affects.

In order to cause Parkinson's Disease, the level of toxic exposure usually has to be either acute or chronic. Mild exposure to some of the known toxic causes would have no noticeable effect.

Some other potentially toxic causes of Parkinson's Disease, such as copper and manganese are actually beneficial to health in normal quantities, and can only cause Parkinson's Disease in very large quantities. For a completely comprehensive, fully referenced, up to date and printable summary of each of the toxic causes of Parkinson's Disease, go to Toxic causes of Parkinson's Disease


Scientists are working to better understand the broad range of environmental exposures linked to Parkinson’s disease. While the exact cause of Parkinson’s disease is unknown, most experts agree that the condition is caused by a combination of genetic and environmental factors (chemicals from occupational exposure or rural living, head trauma, etc.). Some experts explain it this way: genes load the gun and environment pulls the trigger.
We know that exposure to certain environmental factors increases an individual’s risk of developing Parkinson’s disease, but exposure to any one of these factors doesn’t mean a person will actually develop the disease. In most people with Parkinson’s, it’s a collection of factors acting synergistically that ultimately leads to symptoms and a diagnosis.

Non-genetic risk factors for Parkinson’s disease
While age is the biggest risk factor for Parkinson's, many chemicals used in various professions – such as welding, farming, military activities and industrial processes – have been associated with the disease. Some specific chemicals and other factors are listed below:
• Insecticides: permethrin and beta-hexachlorocyclohexane (beta-HCH)
• Herbicides: paraquat and 2,4-dichlorophenoxyacetic acid
• Fungicide: maneb
• Agent Orange used in the Vietnam War
• MPTP, a synthetic neurotoxin (can cause Parkinsonism)
• Head injuries

New Evidence Suggests Parkinson's Might Start in the Gut, Not the Brain

“Now a new study in mice has shown that the toxic fibres that build up around the nerve cells of Parkinson's patients can influence the nerves in the brain in a matter of weeks.
"We have discovered for the first time a biological link between the gut microbiome and Parkinson's disease," said lead researcher Sarkis Mazmanian from the Californian Institute of Technology (Caltech).
This research depicts the findings of Sampson et al., who show that signals from gut microbes are required for the neuroinflammatory responses as well as hallmark gastrointestinal and a-synuclein-dependent motor deficits in a model of Parkinson's disease.
Credit: Sampson et al./Cell 2016


PD is considered a multifactorial disease resulting from the effect of environmental factors and genetic susceptibility. Linking PD to an environmental cause, however, seems difficult. This is because of the long presymptomatic period

Despite the mysterious cause of PD, the pathologic and neurochemical basis of parkinsonian signs and symptoms have been in part unraveled as discussed earlier. Toxic insults could both modify the structure of -synuclein and interfere with the ubiquitin-proteasomal pathway, thus, promoting -synuclein aggregation and impairing the process of degradation of the abnormal protein [29].

Barlow et al. [30] found that even in utero exposure with subsequent genetic damage can be a risk factor for neurodegenerative diseases, for example, Alzheimer and PD. This can explain the finding of Tsai et al. [31] who described what he has called a young onset parkinsonian disease (YOPD) which is a peculiar group of patients developing PD before 40 years on contrary to normal age group of more than 50 years.

The reason for trying to identify toxic causes of PD (besides future protection) is the search for a perfect PD model. Scientists are trying to unravel the exact cause of PD; also we are trying to find out a perfect therapy and both the therapy and cause need a perfect model to act on. Finding a toxin that can be a true cause of PD would solve the problem.

So, what are those toxins that can be candidates for toxic PD models? Many have been described including the following.


Many risk factors have been implicated in PD. Since the discovery of MPTP, parkinsonian inducing effects this arouse the possibility of other similar compounds relevant to MPTP, for example, paraquat to induce PD [32]. Numerous classes of pesticides were introduced during the twentieth century. Although PD existed long before the introduction of these pesticides, the thought is that pesticide exposure has contributed to the increased incidence of the disease [33].


The fumigant class encompasses a variety of agents most commonly used to control insects or fungi in grains, soil, or other various consumables. Fumigants, such as ethylene dibromide, are highly toxic to humans but most adverse actions are nonneuronal [34]. Although there is some evidence that carbon disulfide-based fumigants can induce parkinsonian-like neurotoxicity [35]. This chemical class is not suitable for induction of PD in an animal model.

Fungicides are agents of a wide variety of chemical structures. Maneb, or manganese ethylene bis-dithiocarbamate, is one type with possible parkinsonian symptoms which may be secondary to exposure to the manganese metal core [33].
In fact, maneb is one of the toxins used to induce PD in animal models but usually in combination with other agents specially paraquat [36]. The maneb effect has been undoubtedly documented to increase the severity of PD models which make it one of the good candidates in PD research [37].

Herbicides (Paraquat)
The possibility that paraquat (1,1_-dimethyl-4,4_-bipyridinium) may damage the nigrostriatal dopaminergic system and therefore contribute to the neuropathology of Parkinson’s disease (PD) was first proposed in the mid 1980s following the observation that its chemical structure closely resembles that of MPP+(1-methyl-4-phenylpyridinium ion) [38].
Animal studies confirmed the ability of paraquat to induce selective dopaminergic nigrostriatal degeneration [39–41].
Li et al. [40] have developed subacute model for inducing PD in mice through using intraperitoneal injection of paraquat (10 mg/kg) in old C57/bl mice. Similar results were obtained 2 years later by Kuter et al. [41] using a similar approach.
As described earlier, an improvement in modeling of paraquat induced PD was through adding maneb. A combination which improved the results. It also supported the multiple hit theory of PD [42].
The presence of differences between paraquat and MPTP has been questioned and the answer would come from a group of articles in Toxicological Sciences journal regarding mechanism of paraquat toxicity. Richardson et al. [43] proposed that paraquat would have different mechanisms for toxicity than MPTP. Later, Ramachandiran et al. [44] have shown that paraquat and MPTP have divergent mechanisms of toxicity. Another hot debate emerged on the same journal regarding the possibility of different mechanisms of neurotoxicity between paraquat and MPTP [45–48].
Despite the debate, there are certainly toxicokinetic and toxicodynamic differences which would give paraquat a unique pattern of neurotoxicity. Despite the fact that paraquat models are still less validated than the previous well-established MPTP models [49], combining maneb with paraquat would introduce a promising modeling technique that can be (in our opinion) a better model specially with the limitation in the other available toxic models.

There are several subclasses of insecticides, each with their own subdivisions. Many of the compounds in this class are, by design, neurotoxic. Similarities between the insect and human nervous systems can lead to cross-toxicity of these compounds [33].


Parkinson's Disease Discovered

Parkinson's disease was first characterized extensively by an English doctor, James Parkinson, in 1817. Today, we understand Parkinson's disease to be a disorder of the central nervous system that results from the loss of cells in various parts of the brain, including a region called the substantia nigra. The substantia nigra cells produce dopamine, a chemical messenger responsible for transmitting signals within the brain that allow for coordination of movement. Loss of dopamine causes neurons to fire without normal control, leaving patients less able to direct or control their movement. Parkinson's disease is one of several diseases categorized by clinicians as movement disorders.
The exact cause of Parkinson's disease is unknown, although research points to a combination of genetic and environmental factors. If a continuum existed, with exclusively genetic causes at one end and exclusively environmental causes at the other, different Parkinson's patients would likely fall at many different places along that continuum.

Genetic Causes
In the past 10 years, researchers have identified a number of rare instances where Parkinson's disease appears to be caused by a single genetic mutation. In these cases, the mutated gene is passed from generation to generation, resulting in a great number of Parkinson's disease cases within an extended family. Mutations in the LRRK2 gene are the greatest genetic contributor to Parkinson's disease disovered to date.

Environmental Causes
On the opposite end of the continuum, in the early 1980s, a group of heroin users in California took drugs from a batch contaminated with a substance called MPTP. After ingesting this chemical, the drug users were stricken with a form of Parkinson's disease that was primarily, if not exclusively, "environmental" in origin.

A Combination of Both
For most Parkinson's patients, the cause lies somewhere in the middle. While many Parkinson's patients report one or more family members with the disease, it is not always clear that one or several genes are the cause. Similarly, while some patients suspect that exposure to one or another chemical or environmental toxin caused their Parkinson's disease, this also cannot be conclusively proved. Scientists currently believe that in the majority of cases, genetic and environmental factors interact to cause Parkinson's disease. Research into this subject continues aggressively every day. Unfortunately, however, it is generally impossible to determine what specifically caused an individual's Parkinson's disease.

Other Risk Factors
Because the causes of Parkinson's disease are unknown, there is no scientifically validated preventive course to reduce the risk of its onset. The single biggest risk factor for Parkinson's disease is advancing age. Men have a somewhat higher risk than women.
That being said, a number of studies have highlighted factors that are associated with either greater or lesser risk of Parkinson's disease. For example, smoking and caffeine consumption have been associated with lower rates of Parkinson's disease, while head injury and pesticide exposure have been associated with higher risk. While such studies do not definitively link these factors with Parkinson's disease one way or another, they highlight areas where further research may guide us to risk-prevention or treatment strategies.

What is drug-induced parkinsonism?
A small number of people with parkinsonism have developed their symptoms following treatment with particular medications. This form of parkinsonism is called ‘drug-induced parkinsonism’.
People with idiopathic Parkinson’s disease and other causes of parkinsonism may also develop worsening symptoms if treated with such medication inadvertently.

What drugs cause drug-induced parkinsonism?

Any drug that blocks the action of dopamine (referred to as a dopamine antagonist) is likely to cause parkinsonism. Drugs used to treat schizophrenia and other psychotic disorders such as behaviour disturbances in people with dementia, known asneuroleptic drugs, are possibly the major cause of drug-induced parkinsonism worldwide. Parkinsonism can occur from the use of any of the various classes of neuroleptics.
The atypical neuroleptics – clozapine (Clozaril) and quetiapine (Seroquel), and to a lesser extent olanzapine (Zyprexa) and risperidone (Risperdal) – appear to have a lower incidence of extrapyramidal side effects, including parkinsonism. These drugs are generally best avoided by people with Parkinson’s, although some may be used by specialists to treat symptoms such as hallucinations occurring with Parkinson’s.
For people with Parkinson’s, anti-sickness drugs such as domperidone (Motilium) or ondansetron (Zofran) are the drugs of choice for nausea and vomiting.
As well as neuroleptics, some other drugs can cause drug-induced parkinsonism. These include some medications for dizziness and nausea such as prochlorperazine (Stemetil); and metoclopromide (Maxalon), which is used to stop sickness and in the treatment of indigestion.
Calcium channel blocking drugs used to treat high blood pressure, abnormal heart rhythm, angina pectoris, panic attacks, manic depression and migraine may occasionally cause drug-induced parkinsonism. Calcium channel blocking drugs are, however, widely used to treat angina and high blood pressure, and it is important to note that most common agents in clinical use probably do not have this side effect. These drugs should never be stopped abruptly without discussion with your doctor.
A number of other agents have been reported to cause drug-induced parkinsonism, but clear proof of cause and effect is often lacking. Amiodarone, used to treat heart problems, causes tremor and some people have been reported to develop Parkinson’s-like symptoms. Sodium valproate, used to treat epilepsy, and lithium, used in depression, both commonly cause tremor which may be mistaken for Parkinson’s.

What are the characteristics of drug-induced
parkinsonism and how does it differ from idiopathic Parkinson’s?


Drug-induced parkinsonism is more likely to be symmetrical (on both sides of the body) and less likely to be associated with tremor, although it can sometimes present asymmetrically and with a tremor. Akinesia with loss of arm swing can be the earliest feature. Bradykinesia can be an early common symptom, causing expressionless face, slow initiation of movement and speech difficulties.

If the offending drug is stopped, will the drug-induced parkinsonism improve and if so, how long will this take?
Generally, the majority of people will recover within two months, and often within hours or days, of stopping the offending drug. However, some people may take as long as two years.

Pesticides and herbicides like glyphosate now strongly linked to Parkinson's disease and other neurological disorders

Pesticides and herbicides are destroying the body's natural relationship
This dilemma is born out of mankind's desire to control and manipulate nature by taking chemical shortcuts with agriculture and food production. When will we begin to understand that the myriad of chemicals we develop, spray, breathe, eat and inject into our bodies, is interfering with the natural, healthy relationship between our organ systems? Independent scientists are now beginning to understand that the mechanism that the herbicide glyphosate uses to destroy plants, is the same mechanism that kills the good microbes in the human gut. It's these good microbes that assist in the proper absorption of nutrients, elimination of waste, detoxification of impurities, and protection of the gut lining and blood. Without a strong microbiome, the human body is slow to initiate immune responses to pathogenic stressors.

Genes are being damaged at a young age as infants are inundated with chemicals
Infants are being introduced to pesticides and herbicides at such a young age, exposed through hospital feeding tubes and corn syrup formulas. The burden of chemicals is much more devastating on small, developing bodies which are typically 1/30th the weight of a grown adult. Additionally, the rate at which infants are being injected with vaccine aluminum, antibiotics, formaldehyde, MSG, animal and human body cell lines, and in some cases mercury, suggests that their healthy genes may never see normal conditions to express their innate, healthy disposition.

As these chemicals break down the lining of the gut and deplete the body's intelligent microbiology, the blood is easily infiltrated, allowing chemicals to circulate through the entire system, taxing filtration systems like kidneys, gall bladder, liver and skin. If the body is exposed for long enough, the pesticides, heavy metals and other chemicals may even circulate to the brain, causing neuron cell death and interfering with hormones and mental homeostasis.

Strong links between pesticides, herbicides and Parkinson's

Pesticides and herbicides are sprayed so carelessly today, infiltrating both the chemistry of the food and the air we breathe. Distressingly, farmers are often told to spray cancer-causing glyphosate on a barren field, post-harvest, just to make sure no weeds will grow through the winter. It's so sad.

From the Division of Cancer Epidemiology and Genetics at the National Cancer Institute, researchers found that chronic pesticide exposure is linked directly to Parkinson's disease. Parkinson's represents a cluster of health problems originating in the central nervous system, and is signified by motor function decline, twitching in the extremities, cognitive deficits, and potentially, full-blown dementia.

In May 2013, the American Academy of Neurology (AAN) published results on over 100 studies from around the world, showing that increased exposure to pesticides, herbicides and solvents is "likely associated with a higher risk of developing Parkinson's disease." Farmers that work directly with these chemicals suffer the most from neurological disorders, especially Parkinson's. Pesticide exposure in rural areas was directly linked to a two-fold increased risk of developing Parkinson's.

In a 2011 study led by researchers at the University of California Los Angeles's School of Public Health, simple, routine home-and-garden pesticide exposure resulted in a three times increased risk of Parkinson's. In a meta-analysis, 17 out of 19 studies showed that increased duration of exposure to pesticide and herbicide chemicals was directly linked with increased risk of Parkinson's.

The human body is not strengthened when exposed to industrial chemicals; exposure to pesticides does not make the body tougher. This onslaught of chemicals on the lawn, on our food crops and in the home, is slowly, silently weakening us. The damage is observed and felt all the way down to the hormonal, microbial and genetic level, destroying the body's natural harmony with and within its environment.

Toxic Causes of Parkinson’s Disease


A small proportion of cases have a toxic cause as the sole or a partial cause of Parkinson's Disease. The toxic exposure usually has to be acute or chronic. Symptoms normally develop when the toxic exposure occurs or soon after, or gradually increase over time when the exposure persists. Symptoms do not develop years later as is sometimes claimed. Avoidance of the source of toxicity can lead, in most cases, to a reduction in the symptoms but with some toxins this can take years.



Annonaceae is a family of flowering plants that is also called the custard apple family. Some annonaceae species produce edible fruits. Annonaceae contain acetogenins. In Guadeloupe, Annonaceae are consumed as herbal teas and fruits, especially soursop. Annonacin, which is the most abundant acetogenin, is toxic to dopaminergic neurons in nanomolar concentrations. Acetogenins are potent mitochondrial toxins, like other Parkinsonism inducing compounds. The concentrations of annonacin, the major acetogenin in Annona Muricata, are so high that one fruit or can of nectar per day is more in a year than induced brain lesions in rats receiving purified annonacin by intravenous infusion.

Carbon disulfide is a colourless, flammable, and poisonous liquid with the chemical formula CS2. Carbon disulfide is in pesticides used as fumigants, disulfiram, which is a drug used in the treatment of chronic alcoholism, industrial solvents, and solvents used in the production of viscose rayon and cellophane film. Carbon disulfide interferes with the enzyme pyridoxal 5-phosphate, which is essential for the formation of dopamine from L-dopa. So carbon disulfide might cause Parkinson's Disease symptoms by reducing the formation of L-dopa.


Carbon monoxide is a colourless, odourless, and tasteless gas that is slightly less dense than air. Carbon monoxide is produced from the partial oxidation of carbon containing compounds when there is not enough oxygen to produce carbon dioxide. Common sources are motor vehicle exhaust fumes, and cigarette smoke. It also forms when fuels such as coal, paraffin, oil or wood, and especially natural gas, do not burn completely in appliances such as heaters, furnaces, stoves, water heaters, and ovens. However, carbon monoxide rarely causes Parkinson's Disease. It normally requires the person having gone in to a coma as a result of the carbon monoxide poisoning before symptoms of Parkinson's Disease develop.


Copper, whose chemical symbol is Cu, is a chemical element that is required as an essential nutrient, but which can be toxic in large quantities. Common causes of copper toxicity include Wilson's Disease in which there is a genetic accumulation of copper, copper mines, copper cooking pots, copper plumbing, extreme consumption of nutritional supplements that contain copper. Excessive copper can cause the formation of a copper-dopamine complex, which leads to the oxidation of dopamine to aminochrome.


Cyanide is contained in drugs such as potassium cyanide and sodium cyanide. Cyanides can also be produced by certain bacteria, fungi, and algae, and are found in a number of foods and plants. Cyanide occurs naturally in cassava roots (manioc). Fruits that have a pit, such as cherries or apricots, often contain either cyanides or cyanogenic glycosides in the pit. Bitter almonds, from which almond oil and flavouring is made, also contain cyanide. Hydrogen cyanide is contained in vehicle exhaust and in tobacco smoke. The smoke of burning plastics contains hydrogen cyanide, and so house fires often result in cyanide poisonings. Cyanides are one of the very few soluble compounds of gold and thus are used in electroplating, gold mining, metallurgy and jewellery for chemical gilding, buffing, and extraction of gold. Cyanides and hydrogen cyanide are used in the production of chemicals, photographic development, making plastics, fumigating ships, and some mining processes. Potassium ferrocyanide is used to achieve a blue colour on cast bronze sculptures.


Cycad seeds of the Cycas micronesica contain ss-methylamino-L-alanine (BMAA), which have been implicated as the cause of toxicity due to the involvement of a product of BMAA and a beta-carbamate. Cycad seeds, by different means, can cause Amyotrophic lateral sclerosis (ALS) and Parkinsonism-dementia complex (PDC). This occurs in Guam, which is in the Mariana islands in the Pacific Ocean, the Kii peninsula of Japan, and New Guinea, which is divided between Indonesia and Papua New Guinea.

Dieldrin is used as a pesticide. Dieldrin was the most frequently detected Organochlorine pesticide in people with Parkinson's Disease thereby suggesting that dieldrin may be associated with Parkinson's Disease. Dieldrin levels were above normal in brains of people with Parkinson's Disease. Dieldrin adversely affects cellular processes associated with Parkinson's Disease including mitochondrial function and reactive oxygen species production. Dieldrin showed inhibitory effects on proteasome activities at low concentrations. Dieldrin induces apoptosis in dopaminergic neurons via caspase-3-dependent proteolytic activation of protein kinase C delta.


Hydrocarbons are organic compounds consisting entirely of hydrogen and carbon. Common sources are : hydrocarbon poisoning such as that of benzene and petroleum usually occurs accidentally by their inhalation or ingestion. Sources of hydrocarbons include : natural gas, cooking gas, petrol, gasoline. People that have been subjected to exposure to petroleum products, especially when working with petroleum products, increase the likelihood of developing Parkinson's Disease. Working in a gas station (petrol station) increased the risk of Parkinson's Disease by 2.6 times.


Lead is a chemical element that is toxic in humans. Common sources are : lead contaminated soil, ingestion of lead dust or chips from deteriorating lead-based paints, air pollution from the processing of lead, food grown in contaminated soil, drinking water from plumbing and fixtures that are either made of lead or have trace amounts of lead in them. Lead can also be found in cosmetics in some countries, some herbal remedies, and in toys such as many from China. Serious and chronic exposure to lead can more than double the likelihood of developing Parkinson's Disease. Milder exposure to lead did not increase the likelihood of Parkinson's Disease.


The main active element of Maneb is manganese ethylene-bis-dithiocarbamate. Maneb is used as a fungicide. There is a greatly increased likelihood of developing symptoms by people involved in horticulture and agriculture. As Maneb contains manganese it is possible that it causes Parkinson's Disease symptoms via the same means as manganese, which is by inhibiting tyrosine hydroxylation. Tyrosine hydroxylation is essential for the formation of dopamine. It would thereby lower dopamine levels. The effects of Maneb are potentiated when there is simultaneous exposure to the pesticide Paraquat.


Manganese is a chemical element that is required as a nutrient but can be toxic in large quantities. Occupational exposures occur in welding, when highly exposed, miining as miners are surrounded by manganese dust and airborne manganese particles, alloy production, processing, ferro-manganese operations especially in which manganese ore or manganese compounds are turned into steel, and work with agrochemicals. Areas surrounding manganese heavy industry could also become affected by exposure to manganese. It could occur with very excessive use of manganese supplements. Manganese can cause Manganism, which is a medical disorder that is virtually no different in its symptoms from Parkinson's Disease.


Mercury is a chemical element that is toxic in humans. Common sources are : dietary fish intake, ethnic over-the-counter medications, occupational exposures to mercury vapour, possession of dental amalgam fillings, gold production, skin ointment, and some soaps. One of the chief means of toxicity is via the enzyme pyruvate dehydrogenase (PDH), which is irreversibly inhibited by several mercury compounds. However, the cause of the symptoms of Parkinson's Disease is likely to be due to the fact that mercury potently causes the release of dopamine, thereby lowering dopamine levels.


MPTP (1-methyl 4-phenyl 1,2,3,6-tetrahydropyridine) is a chemical that may be produced accidentally during illicit manufacture of the recreational drug MPPP, which is a synthetic heroin substitute. The most common source is the recreational drug MPPP (Desmethylprodine). It was also developed but unused as a herbicide. It is also an industrial toxin. MPTP inhibits tyrosine hydroxylation, which is essential for the formation of dopamine. So MPTP causes Parkinson's Disease by lowering dopamine levels.


N-Hexane is an alkane. Most of the n-hexane used in industry is mixed with solvents. The major use for solvents containing n-hexane is to extract vegetable oils from crops such as soybeans. These solvents are also used as cleaning agents in the printing, textile, furniture, and shoemaking industries. It is also used by chemists. Certain glues used in the roofing and shoe and leather industries also contain n-hexane. Several consumer products contain n-hexane, such as gasoline, spot removers, quick-drying glues used in various hobbies, and rubber cement.



Nitrogen dioxide is an intermediate in the industrial synthesis of nitric acid, millions of tons of which are produced each year. At higher temperatures it is a reddish-brown gas that has a characteristic sharp, biting odour and is a prominent air pollutant. Exposure of nitrogen dioxide to tyrosine hydroxylase results in the nitration of its tyrosine residues. High exposure to nitrogen dioxide, largely because of pollution, trebled the risk of Parkinson's Disease. Lower exposures did not significantly increase the risk.


Octenol, which is commonly known as mushroom alcohol, is produced by several plants and fungi. Octenol can often be inhaled by humans after being produced in damp, mouldy or water damaged buildings. Octenol interferes with two genes involved in the creation of dopamine : the human plasma membrane dopamine transporter (DAT) and the human VMAT ortholog (VMAT2). This demonstrates that 1-octen-3-ol exerts toxicity via disruption of dopamine homeostasis.


Organophosphorous pesticides are used primarily in pest control as alternatives to chlorinated hydrocarbons that can persist in the environment. Organophosphorous pesticides are used in agriculture. Organophosphorus chemicals have also been used as active ingredients in household pesticides for several decades. Organophosphorus pesticides are significantly associated with Parkinson's Disease. The frequent use of household pesticides that contain organophosphorus chemicals increased the chances of developing Parkinson's Disease by 71% .


Paraquat is used as a herbicide. There is a greatly increased likelihood of developing symptoms by people involved in horticulture and agriculture. People with Parkinson's Disease are more than twice as likely to report heavy exposure to pesticides over their lifetime as family members without Parkinson's Disease. Those people affected were usually those who used a lot of pesticides in their homes and in their hobbies rather than those who routinely used pesticides for their occupation. Pesticides are also known to affect well water. However, it is claimed that Paraquat only potentiates the effect of Maneb, and that it has no effect on humans on its own.


Rotenone is used as an insecticide. There is a greatly increased likelihood of developing symptoms by people involved in horticulture and agriculture. People with Parkinson's Disease are more than twice as likely to report heavy exposure to pesticides over their lifetime as family members without Parkinson's Disease. Those affected were usually those who used a lot of pesticides in their homes, rather than those who routinely used pesticides for their occupation. Insecticides are also known to affect well water. Rotenone is commonly used to treat parasitic mites on chickens and other fowl, and so can be found in poultry. Rotenone inhibits tyrosine hydroxylation, which is essential for the formation of dopamine.


Toluene is used as an octane booster in fuel, as a solvent in paints, paint thinners, chemical reactions, rubber, printing, adhesives, lacquers, leather tanning, disinfectants, producing phenol and TNT. Toluene is also used as a raw material for toluene diisocyanate, which is used in the manufacture of polyurethane foams. Toluene is also used in the manufacture of dyes. Dyes are highly associated with the symptoms of Parkinson's Disease. The means of toxicity is unknown. Toluene has been shown to cause Parkinson's Disease or has been associated with people with Parkinson's Disease.


Trichloroethylene is a solvent with a sweet smell that is a clear non-flammable liquid. Trichloroethylene is a solvent that is used in industry and the military and is a common environmental contaminant. It has been used to extract vegetable oils, in coffee decaffeination, and the preparation of flavouring extracts from hops and spices. The means of toxicity is unknown. Workers with workstations adjacent to the source of trichloroethylene and who were subjected to chronic inhalation and dermal exposure from handling trichloroethylene-soaked metal parts all had Parkinson's Disease.


March 1, 2014 by Editor Leave a Comment


Perhaps everyone has aluminum foil in their kitchens, by the BBQ, or taken with us to the camp site to use on the fire or afterwards to store our cooked food. It has so many uses.
However when it comes to using aluminum in food preparation and storage, what has science informed us? Aluminum foil is safe when used cold, like wrapping a sandwich for the fridge; however, using it hot, or even warm, the foil is degraded and SEEPS into the food.

When hot or heated, the levels of aluminum that leak into the food are considerably higher if the food actually touches the foil, if seasonings are added, or if the food is acidic. The hotter the foil is heated, the more seepage occurs. This result occurs no matter which side of the foil is next to the food.

Aluminum impedes with the digestion of calcium, phosphorus, and fluoride, which can even result in osteoporosis. It harms the kidneys and impairs the liver. It is linked to neurodegenerative diseases like Parkinson’s and Alzheimer’s. It can lead to sleep disturbance, and speech problems.
There are lots of sources of aluminum in our day to day lives; antacids, vaccines, antiperspirants, even drinking water. So we’re already getting a heavy dose of aluminum. Cooking in aluminum foil or aluminum pans increase exposure.


Four most dangerous toxins to the brain (Fluoride #1)
Posted on by Dr. Sharp
(NaturalNews) A healthy mind is a terrible thing to lose, but due to man-made chemicals, the threat of brain function destruction is always present. These are potentially the most offensive perpetrators to the mind, but the good news is that there are easy ways to avoid them.


The decision to add fluoride to most public drinking water systems as a way to fight tooth decay has perhaps had the most insidious and widespread effect on the brains of people across North America.
The Fluoride Action Network (FAN) has reported on a study which found that fluoride is linked to lower IQ, even at the ranges added to U.S. water supplies. In all, FAN said that 34 studies now link fluoride to lower IQ levels in humans, while many other studies link fluoride to learning and memory impairment, fetal brain damage and altered neurobehavioral function.
One study sponsored by UNICEF found that IQ was reduced at just 0.88 mg/l of fluoride, a level that is considered within optimal range and is added to U.S. drinking water systems that serve over 200 million Americans every day.
Removing fluoride from your water supply is often as simple as installing a high-quality water filter.

Heavy metals
Heavy metals are especially sinister toxins that are not easily removed but are commonplace in the bodies and brains of millions thanks to vaccines, dental procedures, agricultural chemicals, industrial pollution, fish, chicken and more.
Those most susceptible to traumatic brain damage due to mercury are newborn babies who have yet to properly develop their blood-brain barrier and are generally subjected to an intense vaccination schedule that injects mercury directly into their body. In babies with a genetic susceptibility, such as a defect in enzymes responsible for detoxifying heavy metals, prenatal and early postnatal exposure to mercury leads to neurological damage resulting in autistic symptoms.
Robert Nash, M.D., also suggests that the toxic effects of mercury can cause a broad spectrum of brain-related diseases including autism, Alzheimer’s, amyotrophic lateral sclerosis, multiple sclerosis, Parkinson’s disease and neurodevelopmental diseases.
To ensure that you don’t endure any future heavy metal poisoning, avoid the flu shot, mercury fillings and chemicals, and take safety precautions when consuming seafood.
Artificial sweeteners
The desire for zero-calorie sweeteners produced the advent of artificial sweeteners, and the negative effects on the brain have been well documented. They are found in sodas, yogurt, chewing gum, cooking sauces, tabletop sweeteners, flavored water, cereals and sugar-free products.
Aspartame is a combination of chemicals, namely aspartic acid (an amino acid with excitatory effects on brain cells), methanol and phenylalanine, and scientists are placing it at the higher end of the range of what is considered toxic. When aspartame breaks down, it produces a compound that is a powerful brain-tumor-causing chemical.
Ninety different symptoms have been documented as a result of aspartame consumption, including anxiety attacks, slurred speech, depression and migraines.
Avoid artificial sweeteners by reading ingredients and avoiding the products above. The FDA also recently approved of calling aspartame AminoSweet, so beware of that ingredient as well.
Monosodium glutamate (MSG)
MSG is a form of concentrated salt added to foods to enhance flavor. It was first used in military rations to give them a better taste, but it was soon adopted by the entire food industry.
Dr. Blaylock, a noted neurosurgeon and authority on excitotoxins such as MSG, discovered that there was silent damage to the brain, and over time he saw destruction of major portions of the brain that could cause Alzheimer’s disease, Parkinson’s and other brain ailments.
The best way to avoid it is to stop eating processed food, or look for it in the ingredients, mainly as monosodium glutamate or yeast extract.

Toxins, Toxins, Toxins seem to be the main cause if not “the” cause of Parkinson’s Dis-ease.
Whether the toxin comes from a pesticide, a vaccine, aluminum foil , artificial sweeteners, MSG or medications, we find ourselves looking at “toxins” again as the cause of this avoidable dis-ease! Avoid the toxins and you probably won’t EVER get this dis-ease of the body. What is nice is that the G2 Sacraments can detox the body of heavy metals, chemicals and pathogens!

What to do to “restore health” for the Parkinson’s Dis-ease
1. Start with the Starting Procedure Protocol G2 to detox:
2. Stop toxins for coming in:
3. A natural diet of REAL food
4. Cannabis oil: Ex-Pofoodlice Capt. with Parkinson’s Disease Astonished by Medical Marijuana Results
Note: if anyone needs guidance with “restoring health” form Parkinson's Dis-ease or ANY dis-ease for that matter please feel free to e-mail me at: This email address is being protected from spambots. You need JavaScript enabled to view it.
Our next Genesis II Church Seminar is at: Amsterdam, Holland July 7th-9th! If interested, please e-mail us at: This email address is being protected from spambots. You need JavaScript enabled to view it.
If you can’t attend a G2 Seminar then the next best way to know ALL the Genesis II Church Sacramental protocols is to do the online course at:

Let’s change the world together!
Archbishop Mark S. Grenon

MMS Saves Lives.





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